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| Reference #: | 1,359 |
| Submit Date: | 05 Nov 2007 |
| Browse Category: | epilepsy |
| Author: | none |
| Email Address: | wsotr@hotmail.com |
| Treatment used: | none |
| You can buy this remedy at: | free |
| Remedy will cost you: | unknown |
| Country of Remedy: | USA |
| Remedy Source: | Waters Singing on the Rocks |
| More Links about this Remedy: | none |
| # Comments posted to this remedy: | 0 |
| Complaints Reported: | 0 |
| # of times remedy read: | 17,132 |
| Dosage Info: | |
| Typical Dosage: | unknown |
| Dosage should be related to weight: | unknown |
| Dosages used in clinical trials are significant: | unknown |
| Maximum dosages in relation to side effects and serious side effects: | unknown |
| Other foods/nutrients/medications that can affect absorption or utilization: | unknown |
| Foods that provide the nutrient recommended as a remedy (or reference giving same): | unknown |
| Ratings: | |
| Total # reviewers: | 0 |
| Average Rating: | 0.00 |
| Effectiveness: | 0.00 |
| No Side Effects: | 0.00 |
| Ease of Use: | 0.00 |
| Effective after long term use: | 0.00 |
| Cost Effectiveness: | 0.00 |
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Vaccinations Increase Risk of Epilepsy This post is very similar to a prior post about vaccinations and the risk of Alzheimer's related to infection with herpes simplex virus. In this post and its citations, a link among epilepsy, herpes simplex, and vaccinations is delineated. .......rationale....... The work of Diane E Griffin and colleagues of Johns Hopkins establishes that measles virus and measles-vaccinations impair cell-mediated immunity (CMI) and increase the likelihood of other viral infections (eg, 1-3). These findings are supported by the work of Martinez et al (i) who admit and are trying to solve vaccination-induced, *long-term* impairment of CMI, and (ii) who mention herpes simplex virus (HSV) as an example of the kind of infection exacerbated by vaccination-induced, long-term impairments of CMI (4). In fact, Martinez et al describe impaired-CMI as a general and long-term effect of traditional vaccinations, not just measles vaccinations (4). Fisher and colleagues have identified HSV as an important component in a high percentage of persons with epileptic seizures (5), a finding supported by a fascinating case history (6). Conclusions: 1. These data suggest that a person's risk of developing HSV-related epileptiform patterns or epilepsy is increased by having had vaccinations and the resulting long-term impairment of cell-mediated immunity. 2. These data also suggest that, if vaccinated, an older person who has a latent infection with one of the herpes-class viruses (i) would be likelier to experience a significant re-activation of the virus, and/or (ii) would be at increased risk for other viral infections -- due to vaccination-induced impairments of cell-mediated immunity. Teresa who believes newer research-findings (eg, 1-4) are helping us understand heretofore unappreciated side-effects of vaccinations. 1) Karp CL et al. Mechanism of suppression of cell-mediated immunity by measles virus. Science. 273(5272):228-31, 1996 Jul 12. The mechanisms underlying the profound suppression of cell-mediated immunity (CMI) accompanying measles are unclear. Interleukin-12 (IL-12), derived principally from monocytes and macrophages, is critical for the generation of CMI. Measles virus (MV) infection of primary human monocytes specifically down-regulated IL-12 production. <2 Hussey GD... Griffin DE. The effect of Edmonston-Zagreb and Schwarz measles vaccines on immune response in infants. Journal of Infectious Diseases. 173(6):1320-6, 1996 Jun. ...Eighty-eight children were immunized at 6 or 9 months of age with the Edmonston-Zagreb (EZ) or Schwarz (SW6, SW9) strain of measles vaccine. Children were studied before and 2 weeks and 3 months after immunization. ...Mitogen-induced lymphoproliferation was decreased at 2 weeks in the SW9 group and at 3 months in all groups and was negatively correlated with measles antibody level at 3 months (r = -.387, P = .003). CD8 T cells, soluble CD8, neopterin, and beta2-microglobulin were increased at 2 weeks in the SW9 group, and soluble CD8 and beta2-microglobulin remained elevated at 3 months. Therefore, measles immunization resulted in suppression of lymphoproliferation, which was most evident in infants with the highest antibody responses and most immune activation. 3) Auwaerter PG... Griffin DE. Changes within T cell receptor V beta subsets in infants following measles vaccination. Clinical Immunology & Immunopathology. 79(2):163-70, 1996 May. Measles produces immune suppression which contributes to an increased susceptibility to other infections. Recently, high titered measles vaccines have been linked to increased long-term mortality among some female recipients.... [The following citation reiterates that vaccinations can impair cell-mediated immunity by shifting cytokines release into a Th2 pattern, thereby allowing intracellular pathogens (eg, many viruses) to be more successful. We note that the authors of this 1997 study are trying to devise a way around the general immune-impairing effect of conventional vaccinations.] 4. Martinez X et al. DNA immunization circumvents deficient induction of T helper type 1 and cytotoxic T lymphocyte responses in neonates and during early life. Proc of the National Academy of Sciences 94.8726-31 1997. ab: The relative deficiency of T helper type 1 (Th1) and cytotoxic T lymphocyte (CTL) responses in early life is associated with an increased susceptibility to infections by intracellular microorganisms. This is likely to reflect a preferential polarization of immature CD4 T cells toward a Th2 rather than a Th1 pattern upon immunization with conventional vaccines... 5. Sanders VJ et al. PRESENCE OF HERPES SIMPLEX DNA IN SURGICAL TISSUE FROM HUMAN EPILEPTIC SEIZURE FOCI DETECTED BY POLYMERASE CHAIN REACTION -PRELIMINARY STUDY. Archives of Neurology. 54(8):954-960, 1997 Aug. Objectives: To determine whether herpes simplex virus causes monofocal epilepsy and to assess the presence of herpes simplex virus 1 (HSV-1) and HSV-2 in surgical specimens from patients with epilepsy by using polymerase chain reaction and Southern blot analysis. Background: Herpes simplex virus is a common neurotropic virus capable of latency within the central nervous system; it has a predilection for the temporal lobe. Central nervous system infection with HSV has been associated with seizure activity. Design and Methods: Surgical specimens were removed from 50 patients as part of a treatment protocol for monofocal epilepsy. Neuropathological classification was done, and adjacent sections were screened for HSV by using polymerase chain reaction. Tissues obtained post mortem from the temporal lobe cortex of persons with Alzheimer disease (n = 17), Parkinson disease (n = 14), or nonneurological disease (n = 17) served as controls. Results: Twenty (40%) of the 50 epilepsy cases and 2 (4%) of the 48 control cases had at least one sample that tested positive for HSV (P < .001). Sixty-seven percent (8/12) of the epilepsy cases with heterotopia were positive for HSV. Conclusions: There was a statistically significant difference in the frequency of HSV-positive surgical specimens from monofocal seizure epicenters compared with nonepilepsy control specimens. These data suggest an association of the virus with seizure activity. All specimens positive for HSV (surgical specimens and control specimens) should be examined to determine the activity or latency state of the virus and cellular localization. 6. Cornford ME, McCormick GF. Adult-onset temporal lobe epilepsy associated with smoldering herpes simplex 2 infection. Neurology 48.425-30 1997. A 40-year-old man with chronic genital herpes simplex infection developed partial complex temporal lobe seizures of insidious onset, with EEG and MRI evidence of a unilateral temporal lobe destructive, atrophic process. Extensive workup did not reveal an infectious etiology. Three years of escalating number and severity of daily seizures with memory loss led to temporal lobectomy. Histologic study revealed active, low-level viral infection in the resected hippocampus and temporal lobe cortex, with immunohistochemical evidence for infection by herpes simplex 2, principally in neurons. In situ hybridization confirmed the presence of herpes simplex virus in neurons. Anticonvulsant-resistant seizure episodes began to recur several times daily soon after surgery, but the addition of acyclovir to the treatment regimen resulted in a substantial reduction in seizure occurrence, maintained for the subsequent 2.5 years. |
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