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Vaccinations Increase Risk of Epilepsy
This post is very similar to a prior post about vaccinations and the risk
of Alzheimer's related to infection with herpes simplex virus. In this
post and its citations, a link among epilepsy, herpes simplex, and
vaccinations is delineated.
The work of Diane E Griffin and colleagues of Johns Hopkins establishes
that measles virus and measles-vaccinations impair cell-mediated immunity
(CMI) and increase the likelihood of other viral infections (eg, 1-3).
These findings are supported by the work of Martinez et al (i) who
admit and are trying to solve vaccination-induced, *long-term* impairment
of CMI, and (ii) who mention herpes simplex virus (HSV) as an example of
the kind of infection exacerbated by vaccination-induced, long-term
impairments of CMI (4). In fact, Martinez et al describe impaired-CMI as a
general and long-term effect of traditional vaccinations, not just measles
Fisher and colleagues have identified HSV as an important component
in a high percentage of persons with epileptic seizures (5), a finding
supported by a fascinating case history (6).
1. These data suggest that a person's risk of developing HSV-related
epileptiform patterns or epilepsy is increased by having had vaccinations
and the resulting long-term impairment of cell-mediated immunity.
2. These data also suggest that, if vaccinated, an older person who has a
latent infection with one of the herpes-class viruses (i) would be
likelier to experience a significant re-activation of the virus, and/or
(ii) would be at increased risk for other viral infections -- due to
vaccination-induced impairments of cell-mediated immunity.
Teresa who believes newer research-findings (eg, 1-4) are helping us
understand heretofore unappreciated side-effects of vaccinations.
1) Karp CL et al. Mechanism of suppression of cell-mediated immunity by
measles virus. Science. 273(5272):228-31, 1996 Jul 12.
The mechanisms underlying the profound suppression of cell-mediated
immunity (CMI) accompanying measles are unclear. Interleukin-12 (IL-12),
derived principally from monocytes and macrophages, is critical for the
generation of CMI. Measles virus (MV) infection of primary human monocytes
specifically down-regulated IL-12 production.
<2 Hussey GD... Griffin DE.
The effect of Edmonston-Zagreb and Schwarz measles vaccines on immune
response in infants.
Journal of Infectious Diseases. 173(6):1320-6, 1996 Jun.
...Eighty-eight children were immunized at 6 or 9 months of age with the
Edmonston-Zagreb (EZ) or Schwarz (SW6, SW9) strain of measles vaccine.
Children were studied before and 2 weeks and 3 months after immunization.
...Mitogen-induced lymphoproliferation was decreased at 2 weeks
in the SW9 group and at 3 months in all groups and was negatively
correlated with measles antibody level at 3 months (r = -.387, P = .003).
CD8 T cells, soluble CD8, neopterin, and beta2-microglobulin were
increased at 2 weeks in the SW9 group, and soluble CD8 and
beta2-microglobulin remained elevated at 3 months. Therefore, measles
immunization resulted in suppression of lymphoproliferation, which was
most evident in infants with the highest antibody responses and most
3) Auwaerter PG... Griffin DE.
Changes within T cell receptor V beta subsets in infants following measles
Clinical Immunology & Immunopathology. 79(2):163-70, 1996 May.
Measles produces immune suppression which contributes to an increased
susceptibility to other infections. Recently, high titered measles
vaccines have been linked to increased long-term mortality among some
[The following citation reiterates that vaccinations can impair
cell-mediated immunity by shifting cytokines release into a Th2 pattern,
thereby allowing intracellular pathogens (eg, many viruses) to be more
successful. We note that the authors of this 1997 study are trying to
devise a way around the general immune-impairing effect of conventional
4. Martinez X et al. DNA immunization circumvents deficient induction of T
helper type 1 and cytotoxic T lymphocyte responses in neonates and during
early life. Proc of the National Academy of Sciences 94.8726-31 1997.
ab: The relative deficiency of T helper type 1 (Th1) and cytotoxic T
lymphocyte (CTL) responses in early life is associated with an increased
susceptibility to infections by intracellular microorganisms. This is
likely to reflect a preferential polarization of immature CD4 T cells
toward a Th2 rather than a Th1 pattern upon immunization with conventional
5. Sanders VJ et al. PRESENCE OF HERPES SIMPLEX DNA IN SURGICAL TISSUE
FROM HUMAN EPILEPTIC SEIZURE FOCI DETECTED BY POLYMERASE CHAIN
REACTION -PRELIMINARY STUDY. Archives of Neurology. 54(8):954-960,
Objectives: To determine whether herpes simplex virus causes monofocal
epilepsy and to assess the presence of herpes simplex virus 1 (HSV-1) and
HSV-2 in surgical specimens from patients with epilepsy by using
polymerase chain reaction and Southern blot analysis.
Background: Herpes simplex virus is a common neurotropic virus capable of
latency within the central nervous system; it has a predilection for the
temporal lobe. Central nervous system infection with HSV has been
associated with seizure activity.
Design and Methods: Surgical specimens were removed from 50 patients as
part of a treatment protocol for monofocal epilepsy. Neuropathological
classification was done, and adjacent sections were screened for HSV by
using polymerase chain reaction. Tissues obtained post mortem from the
temporal lobe cortex of persons with Alzheimer disease (n = 17), Parkinson
disease (n = 14), or nonneurological disease (n = 17) served as controls.
Results: Twenty (40%) of the 50 epilepsy cases and 2 (4%) of the 48
control cases had at least one sample that tested positive for HSV (P <
.001). Sixty-seven percent (8/12) of the epilepsy cases with heterotopia
were positive for HSV.
Conclusions: There was a statistically significant difference in the
frequency of HSV-positive surgical specimens from monofocal seizure
epicenters compared with nonepilepsy control specimens. These data suggest
an association of the virus with seizure activity. All specimens positive
for HSV (surgical specimens and control specimens) should be examined to
determine the activity or latency state of the virus and cellular
6. Cornford ME, McCormick GF. Adult-onset temporal lobe epilepsy
associated with smoldering herpes simplex 2 infection. Neurology 48.425-30
A 40-year-old man with chronic genital herpes simplex infection developed
partial complex temporal lobe seizures of insidious onset, with EEG and
MRI evidence of a unilateral temporal lobe destructive, atrophic process.
Extensive workup did not reveal an infectious etiology. Three years of
escalating number and severity of daily seizures with memory loss led to
temporal lobectomy. Histologic study revealed active, low-level viral
infection in the resected hippocampus and temporal lobe cortex, with
immunohistochemical evidence for infection by herpes simplex 2,
principally in neurons. In situ hybridization confirmed the presence of
herpes simplex virus in neurons. Anticonvulsant-resistant seizure episodes
began to recur several times daily soon after surgery, but the addition of
acyclovir to the treatment regimen resulted in a substantial reduction in
seizure occurrence, maintained for the subsequent 2.5 years.